Oral Pathology Mnemonics Online Course
Weird but effective video mnemonics to help you memorize oral pathology .
This is an comprehensive Oral pathology mnemonics course for dental students and dentists of all skill sets. We cover the better, easier and more fun way of studying oral pathology.
This course is based on the oral pathology lectures I had in dental school.
it covers the most difficult-to-memorize details (difficult to me of course ).
It is not meant to explain the whole oral pathology science (you know, it would take months to do that )
This course helps you to avoid trouble when stuck in exam and your memory starts failing you. It will provide you with a list of techniques and tricks to use to study faster, more efficiently, and with less stress and worry of forgetting.
Most importantly, it will give you an insight of an extremely helpful and reliable technique that will let you overcome any difficulty in remembering difficult stuff.
Plus, you can always build-up upon these mnemonics with all the information you have to know, build-up stories, laugh about it, and never mix-up in the exam
PLUS downloadable PDFs -- if you like using sharpies and highlighters like me :) - *Found as a resource attached to the 1st lecture of each section.
Microglossia
Macroglossia part 1 (introduction)
Macroglossia- part 2 (Bonus: Cretenism is Congenital Hypohyroidism)
Macroglossia part3
Macroglossia - part 4 (Bonus: Acromegaly and Gigantism)
Lingual Thyroid Nodule
Micrognathia
Macrognathia
Hemifacial Hypertrophy
Hemifacial Atrophy
Craniofacial Dyostosis (Crouzen's Syndrome)
Cleido-Cranial Dysplasia (CCD)
Supernumerary teeth
Gemination
concrescence
Dens Invaginatus (dens in dente)
Enamel pearl
Cervical enamel extention
Erosion
Hypoplastic Amelogenesis imperfecta
Hypomaturation Amelogenesis imperfecta
Hypomaturationa and Hypoplastic imperfecta
Genes Associated with Ameologenesis Imperfecta
Dentin Dysplasia
Dentinogenisis-imperfecta
Turners Hypoplasia
Dentin-Dysplasia-final
Intrinsic Staining- Ciprofloxacin
Dry Socket
Focal Sclerosing (condensing) Osteitis
Acute Suppurative Osteomyelitis
Diffuse Sclerosing Osteomyelitis
Chronic Osteomyelitis with Proliferative Periostitis
Osteochemonecrosis - Bisphosphonates
Bonus: Osteomyelitis
Paget's Disease of Bone
Central Giant Cell Granuloma
osteopetrosis
osteogenesis Imperfecta
cherubism
Periapical cemento-osseous dysplasia
Florid Cemento-osseous Dysplasia
Fibrous Dysplasia
Monostotic Fibrous Dysplasia of the Jaws
cherubism
Osteoma
Cemento-Ossifying Fibroma
Hemangioma of Bone
Osteosarcoma
Chondroma and Chondrosarcoma
Multiple Myeloma
Bonus: Classification of Non-odontogenic Tumors of Bone
Langerhans Cell Histiocytosis - part 1
Hand-Schuller-Christian Syndrome -Langerhan's Cell Histiocytosis part 2
Ewing's Sarcoma
Metastatic Tumors in the Jaws
Histiogenic Classification of Odontogenic Cysts
Radicular Cyst
Eruption Cyst
Dentigerous Cyst + Differences between Dentigerous Cyst and Eruption Cyst
Odontogenic Keratocyst (OKC)
Lateral Periodontal Cyst
Glandular Odontogenic Cyst
Solitary Bone Cyst
Aneurysmal Bone Cyst (ABC)
Ameloblastoma - part 1
Ameloblastoma - part 2
Adenomatoid Odontogenic Tumor (AOT)
Calcifying Epithelial Odontogenic Tumor (CEOT)
Calcifying Odontogenic Cyst/Tumor
Squamous Odontogenic Tumor
Ameloblastic Fibroma
Odontogenic Myxoma
Squamous Papilloma
Condylomata lata
Focal Epithelial Hyperplasia
Pyostomatitis Vegetans
Verruciform Xanthoma
Verruca Vulgaris
Giant Cell Fibroma
Epulis Fissuratum
Pyogenic Granuloma
Peripheral Ossifying Fibroma
Fibrous Histiocytoma
Fibromatosis
Lipoma
Neurilemoma
Neurofibroma
Neurofibromatosis Type I (von Recklinghausen’s Disease)
Multiple Endocrine Neoplasia (MEN)
Melanotic Neuroectodermal Tumor of Infancy
Cavernous Lymphangioma
Fibrosarcoma
liposarcoma
angiosarcoma
Kaposi's Sarcoma
Angiolymphoid Hyperplasia with Eosinophilia (ALHE)
Non-Hodgkin’s Lymphoma
Burkitt's Lymphoma
Hodgkin's Lymphoma
Granulocytic Sarcoma (Extra Medullary Myeloid Tumor)
Origins of Benign Salivary gland Tumors
Pleomorphic Adenoma
Warthin's Tumors
Peutz-Jeghers Syndrome
McCune-Albright Syndrome
Drug-induced Pigmentation
Oral Malignant Melanoma
Sturge-Weber Syndrome
Hereditary Hemorrhagic Telangectasia
Leukoedema
White Sponge Nevus
Oral Hairy Leukplakia is associated with Epstein-Barr virus
Oral Hairy Leukoplakia site: tongue-bilaterally
Oral Hairy Leukoplakia: Histology: Presence of koilocyte-like cells
Striated OLP presents with hyperkeratosis and hyperparakeratosis
OLP Histology: Band-like lymphocyte infiltration (mainly CD8 Cells)
OLP Histology: Civatte bodies, Basal Cell Layer Degeneration, Vaculative necrosis
OLP Histology: Saw-tooth Rete Ridges in Skin
OLP is associated with hepatitis C
OLP- Civatte Story wrap-up
OLP Occurs most commonly in older females
OLP has 4 clinical presentations
OLP- Wickham's Striae (skin lesions)
DLE is most common in middle-aged females.
DLE: Skin lesions on face and scalp
DLE causes hair loss
DLE: the oral sites most commonly affected
DLE: Oral clinical presentation
SLE: Skin - Butterfly rash
SLE: Affected oral sites
Premalignant/ potentially malignant Lesions
List of Premalignant Conditions
Conditions/Lesions Caused by Alcohol tobacco
Paterson-Kelly Syndrome: Cause and Features
Leukoplakia diagnosis: by exclusion
Leukoplakia is associated with smoking
Leukoplakia: risk of malignant transformation - Gender
Leukoplakia: risk of malignant transformation- Location
Leukoplakia: risk of malignant transformation: Size
Presence of Candida Albicans increases risk of malignant transformation
Genetic Factors that increase the risk of malignant transformation-1
Genetic Factors that increase the risk of malignant transformation - 2
Genetic Factors that increase the risk of malignant transformation - 3
Radiation dose- how many weeks + days/week + fractions/day.
HSV1-route-of-infection
HSV-1: Incubation period is 1-2 weeks
HSV-1: Primary infection: eruption of small vesicles
HSV-1: primary infection: Herpetic gingivostomatitis + affects any oral site
HSV-1: Primary infection: Symptoms last for 7-10 days
HSV-1: Virus remains latent in the trigeminal ganglion
Start HSV-1: Secondary infection: Vesicles rupture forming ulcers.
Start HSV-1: Secondary infection: Ulcers heal in 1-2 weeks.
HSV-2: Predilection to genital mucosa
HSV-2: remains latent in lambosacral ganglion
HSV-2: Histology: Inclusion bodies within keratinocytes.
Primary Infection= Varicella= chicken pox.
VZV: Route of transmission: airborne, highly contagious.
VZV: incubation period: 2 weeks
Zoster: is an elderly disease
Zoster: follows a dermatomal distribution
Zoster: Ramsay Hunt Syndrome. (latency at cranial nerves VII and VIII)
manifestations are Herpangina, and “Hand, Foot, and Mouth Disease"
HFM disease: affects children
Hand, Foot, and mouth disease: Caused by A16 coxsackie virus
HFM disease: Transmission: airborne and orofecal routes
Herpangina: Transmitted by saliva
Herpangina: affects children
Herpangina: Endemic and seasonal (summer and early autumn)
Herpangina: sites: faucial pillars and soft palate.
Measles: Belongs to paramyxovirus family
Measles: Airborne transmission
Measles: Seasonal (winter and spring).
Measles Virus prodromal symptoms
Measles Virus: Incubation period
Measles Virus: Koplik's spots
The Oral mucosa is mostly affected by 2 types of pemphigus
pemphigus: Reaction occurs at stratum spinosum level's desmosomes
pemphigus: Antibodies mainly IgG
Pemphigus: Antibodies become reactive to desmoglein 3
Pemphigus: Blister formation, no ulcers (Intra epithelial lesions)
Pemphigus: Possible genetic predisposition with certain MHC types..
Pemphigus lesions: First to show, last to go
Pemphigus: Positive Nikolsky's sign
Pemphigus: Histopathology: Tzanck cells (acantholytic cells)
M.M. Pemphigoid: separation of the epithelium from the underlying CT
M.M Pemphigoid: Immunoglobulins (mainly IgG)
MM pemphigoid: reaction to mainly Laminin 5 and BP 180 proteins.
MM Pemphigoid: results in oral ulcers.
MM pemphigoid: Mostly affects adult females
MM Pemphigoid: Positive Nikolsky's Sign
MM Pemphigoid: Outcome: Eyes: Symblepharon, which can lead to blindness.
Bullous Pemphigoid: Patients are 70-80 years
Bullous Pemphigoid: Antigen target is BP 230 and BP 180
Pemphigoid- General: Histopathology: Subepithelial clefting with no acantholysis
Hiba Al- Shawa