Course Description

This is an comprehensive Oral pathology mnemonics course for dental students and dentists of all skill sets. We cover the better, easier and more fun way of studying oral pathology.

  • This course is based on the oral pathology lectures I had in dental school.

  • it covers the most difficult-to-memorize details (difficult to me of course ).

  • It is not meant to explain the whole oral pathology science (you know, it would take months to do that )

 This course helps you to avoid trouble when stuck in exam and your memory starts failing you. It will provide you with a list of  techniques and tricks to use to study faster, more efficiently, and with less stress and worry of forgetting. 

Most importantly, it will give you an insight of an extremely helpful and reliable technique that will let you overcome any difficulty in remembering difficult stuff. 

Plus, you can always build-up upon these mnemonics with all the information you have to know, build-up stories, laugh about it, and never mix-up in the exam

What you'll learn:

  • Oral Pathology Mnemonics: The easy, fun way to study.
  • Recall information quickly in time-tight exams (and pop-quizzes).
  • Weird but very effective tricks that will stay in you brain for a long time. ( = less revising + less stress)
  • Use various memory techniques (Rhymes as, Keyword, Letter and Word, Visual, and Story Mnemonic Strategies)
  • How to recall epidemiology, most commonly affected site of conditions.
  • Remember the other names of a certain condition, even when not-at-all related. (When the exam's question has the other name that you don't recall..oops!)
  • Converting certain numbers (such as genes) to words and sentences that actually make sense.
  • Correlate characteristic facts (eg. Frog egg appearance, Tzanck cells..) and related syndromes to the name of the condition itself.
  • All of this with A LOT of laughter, and "OMG, did chronic osteomyelitis with proliferative periostitis MAGICALLY transform into GARFIELD?!!"


PLUS downloadable PDFs -- if you like using sharpies and highlighters like me :) - *Found as a resource attached to the 1st lecture of each section.

Course curriculum

  • 1

    Developmental Defects of the Tongue

    • Microglossia

    • Macroglossia part 1 (introduction)

    • Macroglossia- part 2 (Bonus: Cretenism is Congenital Hypohyroidism)

    • Macroglossia part3

    • Macroglossia - part 4 (Bonus: Acromegaly and Gigantism)

    • Lingual Thyroid Nodule

  • 2

    Developmental Defects of the jaws

    • Micrognathia

    • Macrognathia

    • Hemifacial Hypertrophy

    • Hemifacial Atrophy

    • Craniofacial Dyostosis (Crouzen's Syndrome)

    • Cleido-Cranial Dysplasia (CCD)

  • 3

    Developmental Alterations in the number of teeth

    • Supernumerary teeth

  • 4

    Developmental Alterations in the Shape of Teeth

    • Gemination

    • concrescence

    • Dens Invaginatus (dens in dente)

    • Enamel pearl

    • Cervical enamel extention

  • 5

    Developmental Alterations in the Structure of Teeth

    • Erosion

    • Hypoplastic Amelogenesis imperfecta

    • Hypomaturation Amelogenesis imperfecta

    • Hypomaturationa and Hypoplastic imperfecta

    • Genes Associated with Ameologenesis Imperfecta

    • Dentin Dysplasia

    • Dentinogenisis-imperfecta

  • 6

    Environmental Alterations of Teeth

    • Turners Hypoplasia

    • Dentin-Dysplasia-final

    • Intrinsic Staining- Ciprofloxacin

  • 7

    Inflammatory Diseases of Bone

    • Dry Socket

    • Focal Sclerosing (condensing) Osteitis

    • Acute Suppurative Osteomyelitis

    • Diffuse Sclerosing Osteomyelitis

    • Chronic Osteomyelitis with Proliferative Periostitis

    • Osteochemonecrosis - Bisphosphonates

    • Bonus: Osteomyelitis

  • 8

    Metabolic Bone Conditions

    • Paget's Disease of Bone

    • Central Giant Cell Granuloma

    • osteopetrosis

    • osteogenesis Imperfecta

    • cherubism

  • 9

    Benign Fibro-osseous Lesions

    • Periapical cemento-osseous dysplasia

    • Florid Cemento-osseous Dysplasia

    • Fibrous Dysplasia

    • Monostotic Fibrous Dysplasia of the Jaws

    • cherubism

  • 10

    Non-odontogenic Tumors of Boneer

    • Osteoma

    • Cemento-Ossifying Fibroma

    • Hemangioma of Bone

    • Osteosarcoma

    • Chondroma and Chondrosarcoma

    • Multiple Myeloma

    • Bonus: Classification of Non-odontogenic Tumors of Bone

  • 11

    Diseases of the Bone

    • Langerhans Cell Histiocytosis - part 1

    • Hand-Schuller-Christian Syndrome -Langerhan's Cell Histiocytosis part 2

    • Ewing's Sarcoma

    • Metastatic Tumors in the Jaws

  • 12

    Cysts of the Oral Region

    • Histiogenic Classification of Odontogenic Cysts

  • 13

    Odontogenic Cysts

    • Radicular Cyst

    • Eruption Cyst

    • Dentigerous Cyst + Differences between Dentigerous Cyst and Eruption Cyst

    • Odontogenic Keratocyst (OKC)

    • Lateral Periodontal Cyst

    • Glandular Odontogenic Cyst

  • 14

    Non-Epithelial Primary Bone Cysts (Pseudocysts)

    • Solitary Bone Cyst

    • Aneurysmal Bone Cyst (ABC)

  • 15

    Odontogenic Tumors

    • Ameloblastoma - part 1

    • Ameloblastoma - part 2

    • Adenomatoid Odontogenic Tumor (AOT)

    • Calcifying Epithelial Odontogenic Tumor (CEOT)

    • Calcifying Odontogenic Cyst/Tumor

    • Squamous Odontogenic Tumor

    • Ameloblastic Fibroma

    • Odontogenic Myxoma

  • 16

    Verrucal & Papillary Lesions

    • Squamous Papilloma

    • Condylomata lata

    • Focal Epithelial Hyperplasia

    • Pyostomatitis Vegetans

    • Verruciform Xanthoma

    • Verruca Vulgaris

  • 17

    Reactive Hyperplastic Fibrous Tissue Lesions

    • Giant Cell Fibroma

    • Epulis Fissuratum

    • Pyogenic Granuloma

    • Peripheral Ossifying Fibroma

  • 18

    Neoplastic Fibrous Tissue Lesions

    • Fibrous Histiocytoma

    • Fibromatosis

  • 19

    Adipose Tissue Lesions

    • Lipoma

  • 20

    Neural Tissue Lesions

    • Neurilemoma

    • Neurofibroma

    • Neurofibromatosis Type I (von Recklinghausen’s Disease)

    • Multiple Endocrine Neoplasia (MEN)

    • Melanotic Neuroectodermal Tumor of Infancy

  • 21

    Vascular Lesions

    • Cavernous Lymphangioma

  • 22

    Soft Tissue Sarcoma

    • Fibrosarcoma

    • liposarcoma

    • angiosarcoma

    • Kaposi's Sarcoma

  • 23

    Reactive Lymphoid Lesions

    • Angiolymphoid Hyperplasia with Eosinophilia (ALHE)

  • 24

    Neoplastic Lymphoid Lesions

    • Non-Hodgkin’s Lymphoma

    • Burkitt's Lymphoma

    • Hodgkin's Lymphoma

    • Granulocytic Sarcoma (Extra Medullary Myeloid Tumor)

  • 25

    Benign Salivary Gland Tumors

    • Origins of Benign Salivary gland Tumors

    • Pleomorphic Adenoma

    • Warthin's Tumors

  • 26

    Oral Pigmented lesions -Syndrome Associated Pigmentation

    • Peutz-Jeghers Syndrome

    • McCune-Albright Syndrome

    • Drug-induced Pigmentation

  • 27

    Oral Pigmented lesions- Increased Number of Melanocytes

    • Oral Malignant Melanoma

  • 28

    Oral Pigmented Lesions - Red and Blue Lesions

    • Sturge-Weber Syndrome

    • Hereditary Hemorrhagic Telangectasia

  • 29

    Hereditary White Lesions

    • Leukoedema

    • White Sponge Nevus

  • 30

    Oral Hairy Leukoplakia |Infectious White Lesions

    • Oral Hairy Leukplakia is associated with Epstein-Barr virus

    • Oral Hairy Leukoplakia site: tongue-bilaterally

    • Oral Hairy Leukoplakia: Histology: Presence of koilocyte-like cells

  • 31

    Oral Lichen Planus | Immune Mediated White Lesions

    • Striated OLP presents with hyperkeratosis and hyperparakeratosis

    • OLP Histology: Band-like lymphocyte infiltration (mainly CD8 Cells)

    • OLP Histology: Civatte bodies, Basal Cell Layer Degeneration, Vaculative necrosis

    • OLP Histology: Saw-tooth Rete Ridges in Skin

    • OLP is associated with hepatitis C

    • OLP- Civatte Story wrap-up

    • OLP Occurs most commonly in older females

    • OLP has 4 clinical presentations

    • OLP- Wickham's Striae (skin lesions)

  • 32

    Discoid Lupus Erythematosus | Immune Mediated White Lesions

    • DLE is most common in middle-aged females.

    • DLE: Skin lesions on face and scalp

    • DLE causes hair loss

    • DLE: the oral sites most commonly affected

    • DLE: Oral clinical presentation

    • SLE: Skin - Butterfly rash

    • SLE: Affected oral sites

  • 33

    Oral Neoplasia and Pre-neoplasia

    • Premalignant/ potentially malignant Lesions

  • 34

    Premalignant Conditions

    • List of Premalignant Conditions

    • Conditions/Lesions Caused by Alcohol tobacco

    • Paterson-Kelly Syndrome: Cause and Features

  • 35

    Leukoplakia | Premalignant Conditions

    • Leukoplakia diagnosis: by exclusion

    • Leukoplakia is associated with smoking

    • Leukoplakia: risk of malignant transformation - Gender

    • Leukoplakia: risk of malignant transformation- Location

    • Leukoplakia: risk of malignant transformation: Size

    • Presence of Candida Albicans increases risk of malignant transformation

    • Genetic Factors that increase the risk of malignant transformation-1

    • Genetic Factors that increase the risk of malignant transformation - 2

    • Genetic Factors that increase the risk of malignant transformation - 3

  • 36

    Treatment of Oral Cancer - Radiation

    • Radiation dose- how many weeks + days/week + fractions/day.

  • 37

    Herpes Simplex Virus-1| Viral Infections | Vesiculo-bullous Disorders

    • HSV1-route-of-infection

    • HSV-1: Incubation period is 1-2 weeks

    • HSV-1: Primary infection: eruption of small vesicles

    • HSV-1: primary infection: Herpetic gingivostomatitis + affects any oral site

    • HSV-1: Primary infection: Symptoms last for 7-10 days

    • HSV-1: Virus remains latent in the trigeminal ganglion

    • Start HSV-1: Secondary infection: Vesicles rupture forming ulcers.

    • Start HSV-1: Secondary infection: Ulcers heal in 1-2 weeks.

  • 38

    Herpes Simplex Virus-2 | Viral Infections | Vesiculo-bullous Disorders

    • HSV-2: Predilection to genital mucosa

    • HSV-2: remains latent in lambosacral ganglion

    • HSV-2: Histology: Inclusion bodies within keratinocytes.

  • 39

    Varicella-Zoster Virus | Viral Infections | Vesiculo-bullous Disorders

    • Primary Infection= Varicella= chicken pox.

    • VZV: Route of transmission: airborne, highly contagious.

    • VZV: incubation period: 2 weeks

    • Zoster: is an elderly disease

    • Zoster: follows a dermatomal distribution

    • Zoster: Ramsay Hunt Syndrome. (latency at cranial nerves VII and VIII)

  • 40

    Coxsackie Virus | Viral Infections | Vesiculo-bullous Disorders

    • manifestations are Herpangina, and “Hand, Foot, and Mouth Disease"

  • 41

    Hand, Foot and, Mouth disease | Viral Infections | Vesiculo-bullous Disorders

    • HFM disease: affects children

    • Hand, Foot, and mouth disease: Caused by A16 coxsackie virus

    • HFM disease: Transmission: airborne and orofecal routes

  • 42

    Herpangina | Viral Infections | Vesiculo-bullous Disorders

    • Herpangina: Transmitted by saliva

    • Herpangina: affects children

    • Herpangina: Endemic and seasonal (summer and early autumn)

    • Herpangina: sites: faucial pillars and soft palate.

  • 43

    Measles |Viral Infection | Vesiculo-bullous Disorders

    • Measles: Belongs to paramyxovirus family

    • Measles: Airborne transmission

    • Measles: Seasonal (winter and spring).

    • Measles Virus prodromal symptoms

    • Measles Virus: Incubation period

    • Measles Virus: Koplik's spots

  • 44

    Pemphigus | Vesiculo-bullous Disorders - Immune mediated

    • The Oral mucosa is mostly affected by 2 types of pemphigus

    • pemphigus: Reaction occurs at stratum spinosum level's desmosomes

    • pemphigus: Antibodies mainly IgG

    • Pemphigus: Antibodies become reactive to desmoglein 3

    • Pemphigus: Blister formation, no ulcers (Intra epithelial lesions)

    • Pemphigus: Possible genetic predisposition with certain MHC types..

    • Pemphigus lesions: First to show, last to go

    • Pemphigus: Positive Nikolsky's sign

    • Pemphigus: Histopathology: Tzanck cells (acantholytic cells)

  • 45

    Mucous Membrane Pemphigoid | Vesiculo-bullous Disorders - Immune mediated

    • M.M. Pemphigoid: separation of the epithelium from the underlying CT

    • M.M Pemphigoid: Immunoglobulins (mainly IgG)

    • MM pemphigoid: reaction to mainly Laminin 5 and BP 180 proteins.

    • MM Pemphigoid: results in oral ulcers.

    • MM pemphigoid: Mostly affects adult females

    • MM Pemphigoid: Positive Nikolsky's Sign

    • MM Pemphigoid: Outcome: Eyes: Symblepharon, which can lead to blindness.

  • 46

    Bullous Pemphigoid | Vesiculo-bullous Disorders - Immune mediated

    • Bullous Pemphigoid: Patients are 70-80 years

    • Bullous Pemphigoid: Antigen target is BP 230 and BP 180

    • Pemphigoid- General: Histopathology: Subepithelial clefting with no acantholysis

Watch Intro Video

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Paget's Disease of Bone

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Cervical Enamel Extension

Watch Intro Video

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Crouzen's Syndrome

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Chronic Osteomyelitis with Proliferative Periostitis

Watch Intro Video

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Microglossia

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E-wing's Sarcoma

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Hand, Foot, and mouth disease: Caused by A16 coxsackie virus

Instructor(s)

Dentist, Educator

Hiba Al- Shawa

Dr. Hiba Al-Shawa is a Dentist with a passion for education, spreading knowledge and making the lives of dental students easier (when possible). She has a special interest in improving the memory through different techniques such as visual and "rhymes as" mnemonic strategies, that she employs in helping students study in a more efficient, fun way.

Reviews on Udemy



Testimonials

Sakshi

"I bought this course a month back as oral pathology was my weakest subject. But after i started reading from this mnemonics course i have actually started loving oral pathology as now i enjoy it. Thanks a lot Dr. Hiba for making all this soo easy and fun for us all. Thank you so much🙏🙏"

Rania

"It is amazing"

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